Abstract

Background and Objectives: The incidence of autism has steadily increased over the last several decades. Yet despite previously published reports and several direct presentations on atypically structured viruses, Public Health officials have seemingly not pursued an infectious cause of autism. This article summarizes the evidence for infection of autistic children with cytopathic viruses that fail to evoke inflammation because of an immune evasion mechanism termed stealth adaptation. These viruses were initially cultured from patients with chronic fatigue syndrome. Certain of these viruses are derivatives of the cytomegaloviruses that infected the species of monkeys used to produce poliovirus vaccines. Community spread of these vaccine-derived viruses can explain the increasing incidence of autism and other chronic illnesses.
Methods: The author reviews the low-stringency Polymerase Chain Reaction (PCR) studies that led to the detection of stealth-adapted viruses and describes the characteristic Cyto Pathic Effects (CPE) caused by these viruses.
Result: Stealth-adapted virus-induced CPE was observed in the culturing of blood samples of eighty percent of approximately five hundred tested children with autism. Positive results were also obtained using cerebrospinal fluid samples.
Interpretation and Conclusion: Autism is viewed as a clinical manifestation of a stealth adapted virus encephalopathy that begins before birth. The increasing prevalence of autism over recent decades is attributed to the use of cells cultured from cytomegalovirus-infected monkeys to produce polio virus vaccines. The monkey cytomegaloviruses can undergo structural changes beyond stealth adaptation, including the incorporation of renegade cellular and microbial genetic sequences. Therapies that are based on enhancing the Alternative Cellular Energy (ACE) pathway should be clinically tested.

Keywords: ACE pathway; Polio vaccines; African green monkey simian cytomegalovirus; SCMV; Rhesus cytomegalovirus; Chronic fatigue syndrome; CFS; PCR