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Abstract

Novel Approaches in Cancer Study

Ethnic Differences in Genetic Immunity to Cancer

  • Open or Close Rumyantsev SN*

    Department of Evolutionary Immunology, USA

    *Corresponding author: Rumyantsev SN, Department of Evolutionary Immunology, USA

Submission: January 19, 2019; Published: January 28, 2019

DOI: 10.31031/NACS.2019.02.000536

ISSN:2637-773X
Volume2 Issue2

Abstract

Main goal of this article is to present more exhaustive information about genetic immunity to cancerous disease. The data about the subsistence of the disease in 125 ethnoses around the World have been analyzed. The groups of most immune and most susceptible ethnoses have been revealed. Causative agent of this disease is a specific eukaryotic genomic parasite, spreading amongst humans via its intrusion in the genomes of susceptible organisms. The Intrusion of cancerous genes is performing by contaminated human gametes, either by the sperm or by the egg. The parasite subsistence of human cancerous disease is functioning at the expense of substances and functions derived from the body of its prey. This trait is cruial for the progression of cancerous disease within a human body but intensive nutrition of developed cancerous tissues leads contaminated person to the loss of his body weight. The subsistence is provided with the possession by cancerous subjects of genetic immunity to the victim’s immune defense and cell regulation. These new notions provide framework and landmarks for the detection and discovery of genomic roots of cancerous disease and encourage new proposals for its healing and prevention, as well as for the discovery of the origin and evolution of cancerous disease.

Keywords: Cancerous disease; Cancerous tissues; Cancerous gametes; Cancerous genealogy; Cell regulation; Embryogenesis; Eukaryotic parasite; Genetic immunity; Genome intrusion; Genomic parasite; Femai cancerous gamete; Healing of cancerous disease; Immune response; Mail cancerous gamete; Parasite invasion of a genome; Ovum; Prevention of cancerous disease; Self-procurement of cancerous disease; Selfish genes; Sexual transmission of cancerous disease; Sperm; Xenogamy

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